Session 1. Cardiac Channelopathies

Co-Chairs: Isabelle Baro and Pascale Guicheney

Katja Odening, University of Bern
Novel gene and peptide-based therapies to restore hERG function in the QT syndromes

Carol Ann Remme, Amsterdam UMC
Pharmacological and genetic approaches to target cardiac sodium channels

Nathalie NEYROUD – Sorbonne Université
Enhancing the cardiac sodium current in channelopathies through targeted gene therapy with the Nav1.5 N-terminal domain

Ivy DICK – University of Maryland Baltimore
Diverse Mechanisms in the Pathogenesis of Calcium Channelopathies

Nada BENYAHYA – Mohammed V University of Rabat
Jervell and Lange-Nielsen Syndrome: Molecular Characterization of Two Moroccan Cases with Homozygous KCNQ1 Mutations

Coffee break & poster session

Session 2. Emerging Topics in Channelopathies 

Chair: Sophie Nicole

Angelika Lampert,  Aachen University Hospital
Decoding Neuropathic Pain: Sodium Channels and Sensory Neuron Subtypes

Mohamed Trebak, University of Pittsburgh
Calcium sensing and ER stress signaling in colorectal cancer

Anissa Bara –  Sophion Bioscience
Expanding Automated Patch Clamp Across Scales and Systems

Artem KONDRATSKYI – Nanion Technologies
What to do if you need to functionally characterize 533 ion channel variants

Dinesh SRINIVASAN – Thryv Therapeutics Inc.
SGK1 Inhibition for Long QT Syndrome and Beyond: A Nodal Strategy for Channelopathies and Cardiac Remodeling

Lunch & poster session

Session 3. Organelle Channelopathies 

Co-chairs: Carol Ann Remme and Hector Valdivia

David Yule, University of Rochester
Human mutations in Inositol 1,4,5-trisphosphate receptors: multiple mechanisms
leading to pathology

Dr. Christian Grimm, University of Oxford
Endolysosomal TRPML channels in lung disease

Coffee break & poster session

Alia SYEDA – Dalhousie University
TRPML1 Governs Lysosomal pH and mTORC1 Signaling to Support Genome Integrity in Prostate Cancer

Yvonne E. KLINGL – Ludwig-Maximilians-Universität
TPC1 activity modulation causes iron overload or deficiency by differentially regulating transferrin uptake and endosomal pH

Débora FALCÓN – University of Seville
Ca²⁺-Sensitive Adenylyl Cyclase 8 as a Key Driver of Orai1 Overexpression Following Myocardial Infarction